The more we know, the less the WuFlu is.
...Importantly, we detected SARS-CoV-2-reactive CD4+ T cells in ∼40%–60% of unexposed individuals, suggesting cross-reactive T cell recognition between circulating “common cold” coronaviruses and SARS-CoV-2....
Yup. Somewhere between 40%-60% of people NOT exposed to WuFlu already have antibodies, probably obtained from having the common cold or another flu.
Meantime, Fauci--looking for a bit of attention what with the riots and all that--is still barking about "MO' COVID".
OK, Tony. Tell us about AIDS killing half the planet again, or whatever.
LOL. Thanks for the cherry picking. Time to set you straight.
ReplyDeleteWhile most acute infections result in the development of protective immunity, available data for human coronaviruses suggest the possibility that substantive adaptive immune responses can fail to occur (Choe et al., 2017, Okba et al., 2019, Zhao et al., 2017) and robust protective immunity can fail to develop (Callow et al., 1990). A failure to develop protective immunity could occur due to a T cell and/or antibody response of insufficient magnitude or durability, with the neutralizing antibody response being dependent on the CD4+ T cell response (Crotty, 2019, Zhao et al., 2016). Thus, there is urgent need to understand the magnitude and composition of the human CD4+ and CD8+ T cell responses to SARS-CoV-2. In sum, the ability to measure and understand the human CD4+ and CD8+ T cell responses to SARS-CoV-2 infection is a major knowledge gap currently impeding COVID-19 vaccine development, interpretation of COVID-19 disease pathogenesis, and calibration of future social distancing pandemic control measures.
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ReplyDeleteMy, my, my.
ReplyDeleteIn the first sentence of your rant, you write "....suggest the possibility..."
Damn near anything's possible, of course.
So I suggest the possibility that you are a fake-health Nazi.